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#5
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| Kumar wrote: "Tongue color usually dark "beefy" red o Pale, if caused by pernicious anemia o Fiery red, if caused by deficiency of B vitamins http://www.nlm.nih.gov/medlineplus/e...cle/001053.htm " Can you tell about about difference in three type of above symptms? Today, we may look many people esp. young with Fiery red tongue(lips also). |
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#4
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| monty1945[at]lycos.com wrote: - quote - > Kumar:
Good though. To me, it looks that what goes on in digestive tract it> > I would just like to know if you ever considered the possibility that a > simple dietary change could render all of this "research" obsolete, > because if it is the case that a diet rich in certain kinds of fatty > acids and also oxidized cholesterol is to blame, then why bother with > all these claims about "markers," "endpoints," "associations," etc.? > If the molecular-level evidence is clear, which it is, why not at least > try to follow up on the dietary implications of it? I have for several > years now, and the results are amazing. When I was younger, I never > thought a dietary change could cause my chronic problems to vanish, and > if this did not occur, I doubt that I would still be posting here. I > was trained in grad. school not to waste my time with minor > differences, but the differences are so great (and the evidence so > clear) that I continue to try and reach people like yourself. > > Visit my web site if you decide that a fresh look at the evidence is > worth your time: > > http://groups.msn.com/TheScientificDebateForum- bit less attended currently. But today most may not care for natural healing but may want quick results, though may come out to be harmful in long term. On other side, some risk alike "shocking effect" may be involved in other types of healings than modern healings...as nature may support "survival of fittest". It is upto us what to follow. I feel a via-media may be possible. |
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#3
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| Kumar: I would just like to know if you ever considered the possibility that a simple dietary change could render all of this "research" obsolete, because if it is the case that a diet rich in certain kinds of fatty acids and also oxidized cholesterol is to blame, then why bother with all these claims about "markers," "endpoints," "associations," etc.? If the molecular-level evidence is clear, which it is, why not at least try to follow up on the dietary implications of it? I have for several years now, and the results are amazing. When I was younger, I never thought a dietary change could cause my chronic problems to vanish, and if this did not occur, I doubt that I would still be posting here. I was trained in grad. school not to waste my time with minor differences, but the differences are so great (and the evidence so clear) that I continue to try and reach people like yourself. Visit my web site if you decide that a fresh look at the evidence is worth your time: http://groups.msn.com/TheScientificDebateForum- |
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#2
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| monty1945[at]lycos.com wrote: - quote - > What nation are the two of you from? Aren't you aware that those on
Sorry, you felt like that. Pls consider my thoght as one probable> diets very rich in saturated fatty acids (such as coconut oil) but very > low in unsaturated fatty acids and oxidized cholesterol have hardly any > diabetes, heart disease, etc? > > Do you realize that on such a diet, there would be no "inflammatory" > problems? Do you know that SFAs cannot be made into the inflammatory > molecules in question (such as LTB4) but that the common dietary omega > 6 PUFA, linoleic, is used for this (and that Western diets are now very > rich in linoleic acid)? > > After reading your posts for a while now, I suspect that you have > closed your mind, Kumar, to undeniable, unquestioned, molecular-level > evidence that demonstrates clearly what is occurring. My only interest > is why people such as yourself have closed your minds. After all, this > is antithetical to the scientific method, and you are posting on a > newsgroup that is supposed to be based upon science. possible reason to indicated effect due to systemic inflammation. There can be other reasons. Can ESR/CRP be elevated due to cell's swellings by hypotonicity of blood showing PCV near to lower normal range? If yes, can it effect blod flow to some parts? |
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#1
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| What nation are the two of you from? Aren't you aware that those on diets very rich in saturated fatty acids (such as coconut oil) but very low in unsaturated fatty acids and oxidized cholesterol have hardly any diabetes, heart disease, etc? Do you realize that on such a diet, there would be no "inflammatory" problems? Do you know that SFAs cannot be made into the inflammatory molecules in question (such as LTB4) but that the common dietary omega 6 PUFA, linoleic, is used for this (and that Western diets are now very rich in linoleic acid)? After reading your posts for a while now, I suspect that you have closed your mind, Kumar, to undeniable, unquestioned, molecular-level evidence that demonstrates clearly what is occurring. My only interest is why people such as yourself have closed your minds. After all, this is antithetical to the scientific method, and you are posting on a newsgroup that is supposed to be based upon science. |
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| pls let me know this reply is in responce to what i would also like to participate in discussions like these kind regards Kumar wrote: - quote - > Hello, > > It was discussed that there can be VAT mediated systemic inflammations > due to release of inflamatory cytokines by VAT(visceral adipose > tissues). As such, markers of inflammation i.e.ESR,CRP etc. can remain > elevated in diabetic2 with indulin resistance. > > However diabetics with persisting hyperglycemia may also retain more > water in blood which may cause hypotonicity of blood resulting into > possible cells/erythrocyte's swelling. PCV may also be effected due to > changed tonicity of blood. > > As such, whether such cell's swelling due to hyperglycemia oriented > retention of water in blood can happen ans can such cell swelings also > raise ESR and CRP readings? > > Can such cell swelling also cause vascular inflammation and decreased > supply to tissues? > > Best wishes. |
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#-1
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| Hello, It was discussed that there can be VAT mediated systemic inflammations due to release of inflamatory cytokines by VAT(visceral adipose tissues). As such, markers of inflammation i.e.ESR,CRP etc. can remain elevated in diabetic2 with indulin resistance. However diabetics with persisting hyperglycemia may also retain more water in blood which may cause hypotonicity of blood resulting into possible cells/erythrocyte's swelling. PCV may also be effected due to changed tonicity of blood. As such, whether such cell's swelling due to hyperglycemia oriented retention of water in blood can happen ans can such cell swelings also raise ESR and CRP readings? Can such cell swelling also cause vascular inflammation and decreased supply to tissues? Best wishes. |
| Tags |
| diabetes, inflammation |
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